Publikationen von Rolf Sprengel
Alle Typen
Zeitschriftenartikel (246)
1.
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Dentate gyrus is needed for memory retrieval. Molecular Psychiatry, S. 1 - 12 (2024)
2.
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71 (12), S. 2770 - 2781 (2023)
Increased membrane Ca(2+) permeability drives astrocytic Ca(2+) dynamics during neuronal stimulation at excitatory synapses. Glia 3.
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26 (11), 108116, S. 1 - 24 (2023)
Distinct effects of AMPAR subunit depletion on spatial memory. iScience 4.
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26 (11), 108050, S. 1 - 22 (2023)
Pre- and postsynaptic N-methyl-D-aspartate receptors are required for sequential printing of fear memory engrams. iScience 5.
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28 (2), S. 579 - 587 (2023)
Glutamatergic dysfunction leads to a hyper-dopaminergic phenotype through deficits in short-term habituation: a mechanism for aberrant salience. Molecular Psychiatry 6.
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48 (4), S. 307 - 314 (2022)
Reinforcement rate and the balance between excitatory and inhibitory learning: insights from deletion of the GluA1 AMPA receptor subunit. Journal of Experimental Psychology: Animal Learning and Cognition 7.
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496, S. 190 - 204 (2022)
Increasing the excitatory drive rescues excitatory/inhibitory imbalance and mismatch negativity deficit caused by parvalbumin specific GluA1 deletion. Neuroscience 8.
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16, 908366, S. 1 - 14 (2022)
Comparative severity assessment of genetic, stress-based, and pharmacological mouse models of depression. Frontiers in Behavioral Neuroscience 9.
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16, 877094, S. 1 - 17 (2022)
Phenotyping young GluA1 deficient mice - a behavioral characterization in a genetic loss-of-function model. Frontiers in Behavioral Neuroscience 10.
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12, 102, S. 1 - 13 (2022)
Distinct contributions of GluA1-containing AMPA receptors of different hippocampal subfields to salience processing, memory and impulse control. Translational Psychiatry 11.
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12, 750106, S. 1 - 12 (2021)
Cre-activation in ErbB4-positive neurons of floxed Grin1/NMDA receptor mice is not associated with major behavioral impairment. Frontiers in Psychiatry 12.
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11, 588 , S. 1 - 17 (2021)
Deletion of AMPA receptor GluA1 subunit gene (Gria1) causes circadian rhythm disruption and aberrant responses to environmental cues. Translational Psychiatry 13.
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26, S. 6482 - 6504 (2021)
Imbalanced post- and extrasynaptic SHANK2A functions during development affect social behavior in SHANK2-mediated neuropsychiatric disorders. Molecular Psychiatry 14.
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32 (2), 956797620960392, S. 204 - 217 (2021)
Dissociating representations of time and number in reinforcement-rate learning by deletion of the GluA1 AMPA receptor subunit in mice. Psychological Science 15.
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4, 59 (2021), S. 1 - 21 (2021)
Voltage-independent GluN2A-type NMDA receptor Ca2+ signaling promotes audiogenic seizures, attentional and cognitive deficits in mice. Communications Biology 16.
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228 (1), 113206, S. 1 - 7 (2021)
The GluA1 AMPAR subunit is necessary for hedonic responding but not hedonic value in female mice. Physiology & Behavior 17.
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22 (1), 59, S. 1 - 26 (2020)
Glutamatergic dysfunction and synaptic ultrastructural alterations in schizophrenia and autism spectrum disorder: evidence from human and rodent studies. International Journal of Molecular Sciences 18.
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14, 115, S. 1 - 10 (2020)
Alternative anesthesia of neonatal mice for global rAAV delivery in the brain with non-detectable behavioral interference in adults. Frontiers in Behavioral Neuroscience 19.
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11, 3240 (2020), S. 1 - 16 (2020)
Astrocytic Ca2+ signaling is reduced during sleep and is involved in the regulation of slow wave sleep. Nature Communications 20.
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26, S. 11 - 15 (2020)
Heterogenität SHANK2-bedingter neuropsychiatrischer Störungen. Biospektrum