Publikationen von Christoph Peter

Chevessier, F.; Peter, C.; Mersdorf, U.; Girard, E.; Krejci, E.; McArdle, J. J.; Witzemann, V.: A new mouse model for the slow-channel congenital myasthenic syndrome induced by the AChR εL221F mutation. Neurobiology of Disease 45 (3), S. 851 - 861 (2012)

Pacifici, P. G.; Peter, C.; Yampolsky, P.; Koenen, M.; McArdle, J. J.; Witzemann, V.: Novel mouse model reveals distinct activity-dependent and -independent contributions to synapse development. PLoS One 6 (1), e16469, S. 1 - 13 (2011)

Koenen, M.; Peter, C.; Villarroel, A.; Witzemann, V.; Sakmann, B.: Acetylcholine receptor channel subtype directs the innervation pattern of skeletal muscle. EMBO Reports 6 (6), S. 570 - 576 (2005)

Peter, C.; Korngreen, A.; Witzemann, V.: Mutation of single murine acetylcholine receptor subunits reveals differential contribution of P121 to acetylcholine binding and to channel opening. Pflügers Archiv: European Journal of Physiology 450 (3), S. 178 - 184 (2005)

Peter, C.; Korngreen, A.; Jin, T. E.; Girard, E.; Chevessier, F.; Casas, M.; Krejci, E.; Jaimovich, E.; Witzemann, V.; Molgo, J.: Analysis of the neuromuscular junction, synaptic transmission, and muscle contraction in a knock-in mouse with a slow channel congenital myasthenic syndrome. International Congress (2009)

Peter, C.: Modulation der Aktivität des nikotinischen Acetylcholinrezeptors in transgenen Tiermodellen. Dissertation, 140 S., Ruprecht-Karls-Universität Heidelberg, Heidelberg (2003)